Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. On the flip side, our ability to rescue SERT functionality in KI midbrain synaptoneurosomes with the inhibition of FAK implies elevated FAK signaling downstream of the Pro32Pro33 mutant, as verified by increased pFAK localization in five-HT https://pro3304603.p2blogs.com/31466381/the-best-side-of-pro33